I am sure you know that I love to learn and teach, thus every morning starts with reviewing papers. And I love when medical/research papers enforce or validate or support and strengthen my knowledge.
Being a part of health and wellness company, it’s not that I promote my mitochondrial boosters or NRF2 activators, no, I am on the path of discovering “all of them” and promoting “all of them”. Today’s paper on resveratrol and curcumin, published in the November issue of Clinical Interventions in Aging is one of those validators.
I love to teach and spread the word of healthy living, so please eat your blueberries, and cook with turmeric.
And on top of it: it’s oxidative stress reducer, NAD and sirtuin boost, NRF2 activator and NFKB reducer-AWESOME.
..”In dark chocolate, resveratrol blends nicely with other antioxidants and also minerals, such as iron, copper and manganese. Blueberries don’t have quite as much resveratrol as grapes, but they are also a great source of other antioxidants, dietary fiber, vitamins C and K and manganese…”Please enjoy my latest Facebook post from one of my “morning update”:
From Clinical Interventions in Aging, Nov 2019 issue:
Recovery Of Bone And Muscle Mass In Patients With Chronic Kidney Disease And Iron Overload On
Hemodialysis And Taking Combined Supplementation With Curcumin And Resveratrol:
..”Resveratrol can have several protective effects against age related disorders, including kidney diseases, through the activation of NAD dependent deacetylase and Sirtuin1. SIRT1 deacetylates several substrates and is an important regulator of a wide variety of cellular processes, including stress responses, cell survival, mitochondrial biogenesis and metabolism, occur in response to cellular energy levels and the redox state.
Regarding Turmeric, a study by Shain et al (2016) in Wistar rats treated with 100 mg/Kg/day and exercise for 6 weeks showed that Turmeric helps prevent muscle wasting by regulating NFKB and Nuclear factor erythroid (Nrf2). Furthermore, by deacetylating target proteins SIRT1
can regulate multiple cellular functions, including apoptosis, mitochondrial biogenesis, inflammation, glucose/lipid metabolism, autophagy and cellular stress adaptations. An excess of ROS plays a role in a variety of diseases, as well as in the aging process, which involves numerous cellular response pathways…”